Bulbar cranial nerves. Bulbar and pseudobulbar paresis
Bulbar Cranial Nerves
The bulbar cranial nerves arise from the medulla oblongata and include:
- CN IX – Glossopharyngeal nerve
- CN X – Vagus nerve
- CN XI – Accessory nerve
- CN XII – Hypoglossal nerve
These nerves are responsible for swallowing, phonation, articulation, tongue movements, and palate function.
Anatomy and Physiology
CN IX (Glossopharyngeal)
- Sensation from posterior 1/3 of tongue
- Taste from posterior 1/3 of tongue
- Afferent limb of gag reflex
- Assists swallowing
CN X (Vagus)
- Motor innervation of soft palate, pharynx, and larynx
- Efferent limb of gag reflex
- Controls phonation and swallowing
CN XI (Accessory)
- Sternocleidomastoid muscle
- Trapezius muscle
CN XII (Hypoglossal)
- Motor innervation of tongue muscles
- Controls tongue movements
Examination
1. Soft Palate and Uvula: Ask the patient to say "Ahhh".
- Normal: Symmetrical elevation of soft palate. Uvula remains in the midline
2.Gag Reflex
- Afferent: CN IX. Efferent: CN X
3.Voice Assessment
- Look for: Hoarseness. Nasal speech. Dysarthria.
4. Swallowing
- Assess for: Dysphagia. Choking. Nasal regurgitation
5.Tongue Examination (CN XII)
- Inspect for: Atrophy. Fasciculations. Ask patient to protrude tongue.
6.Accessory Nerve (CN XI): Shoulder shrug against resistance. Head rotation against resistance
Bulbar Paresis (Bulbar Palsy)
Definition: A lower motor neuron (LMN) lesion affecting the nuclei or nerves IX, X, XI, and XII in the medulla.
Causes: Motor neuron disease (ALS). Brainstem infarction. Syringobulbia. Poliomyelitis. Brainstem tumors
Clinical Features: Dysarthria. Dysphagia. Nasal speech. Weak cough. Nasal regurgitation. Tongue atrophy. Tongue fasciculations. Absent gag reflex. Palatal weakness
Pseudobulbar Paresis
Definition: An upper motor neuron (UMN) lesion due to bilateral damage of the corticobulbar pathways.
Causes: Bilateral stroke. Multiple sclerosis. Motor neuron disease (UMN component). Diffuse cerebral disease
Clinical Features: Dysarthria. Dysphagia. Slow, spastic speech. Brisk jaw jerk reflex. Emotional lability (pathological laughing or crying). Increased gag reflex. No tongue fasciculations. No significant tongue atrophy.